Publicacións en colaboración con investigadores/as de Complexo Hospitalario Universitario de Santiago (52)

2022

  1. Concurrent Akt, ERK1/2 and AMPK Activation by Obestatin Inhibits Apoptotic Signaling Cascades on Nutrient-Deprived PC12 Cells

    Cellular and Molecular Neurobiology, Vol. 42, Núm. 5, pp. 1607-1614

  2. Health related quality of life following open versus minimally invasive total gastrectomy for cancer: Results from a randomized clinical trial

    European Journal of Surgical Oncology, Vol. 48, Núm. 3, pp. 553-560

2021

  1. Correction to: Open versus minimally invasive total gastrectomy after neoadjuvant chemotherapy: results of a European randomized trial (Gastric Cancer, (2021), 24, 1, (258-271), 10.1007/s10120-020-01109-w)

    Gastric Cancer

  2. Obestatin signalling counteracts glucocorticoid-induced skeletal muscle atrophy via NEDD4/KLF15 axis

    Journal of Cachexia, Sarcopenia and Muscle, Vol. 12, Núm. 2, pp. 493-505

  3. Open versus minimally invasive total gastrectomy after neoadjuvant chemotherapy: results of a European randomized trial

    Gastric Cancer, Vol. 24, Núm. 1, pp. 258-271

2018

  1. Erratum to: Obestatin controls skeletal muscle fiber-type determination (Scientific Reports, (2017), 7, 1, (2137), 10.1038/s41598-017-02337-4)

    Scientific Reports

  2. Improvement of Duchenne muscular dystrophy phenotype following obestatin treatment

    Journal of Cachexia, Sarcopenia and Muscle, Vol. 9, Núm. 6, pp. 1063-1078

  3. Scrotal Peripheral Primitive Neuroectodermal Tumor

    Current Urology, Vol. 12, Núm. 1, pp. 50-53

2017

  1. Obestatin Increases the Regenerative Capacity of Human Myoblasts Transplanted Intramuscularly in an Immunodeficient Mouse Model

    Molecular Therapy, Vol. 25, Núm. 10, pp. 2345-2359

  2. Obestatin controls skeletal muscle fiber-type determination

    Scientific Reports, Vol. 7, Núm. 1

  3. Obestatin controls the ubiquitin–proteasome and autophagy–lysosome systems in glucocorticoid-induced muscle cell atrophy

    Journal of Cachexia, Sarcopenia and Muscle, Vol. 8, Núm. 6, pp. 974-990

2016

  1. Congenital neurogenic muscular atrophy in megaconial myopathy due to a mutation in CHKB gene

    Brain and Development, Vol. 38, Núm. 1, pp. 167-172

  2. Distinct phosphorylation sites on the ghrelin receptor, GHSR1a, establish a code that determines the functions of ß-arrestins

    Scientific Reports, Vol. 6

  3. Erratum: Corrigendum to “Congenital neurogenic muscular atrophy in megaconial myopathy due to a mutation in CHKB gene” (Brain and Development (2016) 38(1) (167–172) (S0387760415001023) (10.1016/j.braindev.2015.05.008))

    Brain and Development

  4. The role of the obestatin/GPR39 system in human gastric adenocarcinomas

    Oncotarget, Vol. 7, Núm. 5, pp. 5957-5971

  5. β-Arrestin scaffolds and signaling elements essential for the obestatin/GPR39 system that determine the myogenic program in human myoblast cells

    Cellular and Molecular Life Sciences, Vol. 73, Núm. 3, pp. 617-635

2015

  1. Action of obestatin in skeletal muscle repair: Stem cell expansion, muscle growth, and microenvironment remodeling

    Molecular Therapy, Vol. 23, Núm. 6, pp. 1003-1021

2014

  1. Exome sequencing identifies a CHKB mutation in Spanish patient with Megaconial Congenital Muscular Dystrophy and mtDNA depletion

    European Journal of Paediatric Neurology, Vol. 18, Núm. 6, pp. 796-800

2012

  1. The NMR Structure of Human Obestatin in Membrane-Like Environments: Insights into the Structure-Bioactivity Relationship of Obestatin

    PLoS ONE, Vol. 7, Núm. 10

  2. The obestatin/GPR39 system is up-regulated by muscle injury and functions as an autocrine regenerative system

    Journal of Biological Chemistry, Vol. 287, Núm. 45, pp. 38379-38389