Instituto de Investigación Sanitaria de Santiago de Compostela (IDIS)
Research center
Heinrich Heine University Düsseldorf
Düsseldorf, AlemaniaPublications in collaboration with researchers from Heinrich Heine University Düsseldorf (20)
2022
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An International Perspective on Preceding Infections in Guillain-Barré Syndrome: The IGOS-1000 Cohort
Neurology, Vol. 99, Núm. 12, pp. E1299-E1313
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Electrodiagnosis of Guillain-Barre syndrome in the International GBS Outcome Study: Differences in methods and reference values
Clinical Neurophysiology, Vol. 138, pp. 231-240
2021
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Aetiology of acute respiratory infection in preschool children requiring hospitalisation in Europe-results from the PED-MERMAIDS multicentre case-control study
BMJ Open Respiratory Research, Vol. 8, Núm. 1
2019
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Development of a new largely scalable in vitro prion propagation method for the production of infectious recombinant prions for high resolution structural studies
PLoS Pathogens, Vol. 15, Núm. 10
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Proteomic studies reveal disrupted in schizophrenia 1 as a player in both neurodevelopment and synaptic function
International Journal of Molecular Sciences, Vol. 20, Núm. 1
2018
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Regional variation of Guillain-Barré syndrome
Brain, Vol. 141, Núm. 10, pp. 2866-2877
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Research update for articles published in EJCI in 2016
European Journal of Clinical Investigation, Vol. 48, Núm. 10
2017
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International Guillain-Barré Syndrome Outcome Study: protocol of a prospective observational cohort study on clinical and biological predictors of disease course and outcome in Guillain-Barré syndrome
Journal of the Peripheral Nervous System, Vol. 22, Núm. 2, pp. 68-76
2016
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Acute stimulation of brain mu opioid receptors inhibits glucose-stimulated insulin secretion via sympathetic innervation
Neuropharmacology, Vol. 110, pp. 322-332
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The Priority position paper: Protecting Europe's food chain from prions
Prion
2015
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Discovery and Fine-Mapping of Glycaemic and Obesity-Related Trait Loci Using High-Density Imputation
PLoS Genetics, Vol. 11, Núm. 7
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Mudd's disease (MAT I/III deficiency): A survey of data for MAT1A homozygotes and compound heterozygotes
Orphanet Journal of Rare Diseases, Vol. 10, Núm. 1
2014
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Loss of Prion Protein Leads to Age-Dependent Behavioral Abnormalities and Changes in Cytoskeletal Protein Expression
Molecular Neurobiology, Vol. 50, Núm. 3, pp. 923-936
2012
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Human and rat brain lipofuscin proteome
Proteomics, Vol. 12, Núm. 15-16, pp. 2445-2454
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Proteomic, genomic and translational approaches identify CRMP1 for a role in schizophrenia and its underlying traits
Human Molecular Genetics, Vol. 21, Núm. 20, pp. 4406-4418
2009
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Oligomer assembly of the C-terminal DISC1 domain (640-854) is controlled by self-association motifs and disease-associated polymorphism S704C
Biochemistry, Vol. 48, Núm. 32, pp. 7746-7755
2008
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Insolubility of disrupted-in-schizophrenia 1 disrupts oligomer-dependent interactions with nuclear distribution element 1 and is associated with sporadic mental disease
Journal of Neuroscience, Vol. 28, Núm. 15, pp. 3839-3845
2007
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APC Inactivation Associates With Abnormal Mitosis Completion and Concomitant BUB1B/MAD2L1 Up-Regulation
Gastroenterology, Vol. 132, Núm. 7, pp. 2448-2458
2002
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Distant collaboration in drug discovery: The LINK3D project
Journal of Computer-Aided Molecular Design, Vol. 16, Núm. 11, pp. 809-818
1999
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A limited repertoire of mutations of the luteinizing hormone (LH) receptor gene in familial and sporadic patients with male LH-independent precocious puberty
Journal of Clinical Endocrinology and Metabolism, Vol. 84, Núm. 3, pp. 1136-1140