Publicacións en colaboración con investigadores/as de Pennsylvania State University (17)

2023

  1. Phage-Defense Systems Are Unlikely to Cause Cell Suicide

    Viruses

  2. Ribosome inactivation by Escherichia coli GTPase RsgA inhibits T4 phage

    Frontiers in Microbiology, Vol. 14

2022

  1. CRISPR-Cas Controls Cryptic Prophages

    International Journal of Molecular Sciences, Vol. 23, Núm. 24

  2. The role of PemIK (PemK/PemI) type II TA system from Klebsiella pneumoniae clinical strains in lytic phage infection

    Scientific Reports, Vol. 12, Núm. 1

  3. What are the options for treating infections by persister-forming pathogens?

    Environmental Microbiology, Vol. 24, Núm. 10, pp. 4500-4504

  4. What is the fate of the biofilm matrix?

    Environmental Microbiology, Vol. 24, Núm. 10, pp. 4495-4499

2021

  1. Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1

    Autophagy, Vol. 17, Núm. 1, pp. 1-382

2020

  1. (p)ppGpp and its role in bacterial persistence: New challenges

    Antimicrobial Agents and Chemotherapy, Vol. 64, Núm. 10

  2. Mechanisms of tolerance and resistance to chlorhexidine in clinical strains of Klebsiella pneumoniae producers of carbapenemase: Role of new type II toxin-antitoxin system, PemIK

    Toxins, Vol. 12, Núm. 9

2019

  1. Quorum sensing systems and persistence

    Implication of Quorum Sensing System in Biofilm Formation and Virulence (Springer Singapore), pp. 17-27

  2. Relationship between quorum sensing and secretion systems

    Frontiers in Microbiology, Vol. 10, Núm. JUN

  3. Toxins of toxin/antitoxin systems are inactivated primarily through promoter mutations

    Journal of Applied Microbiology, Vol. 127, Núm. 6, pp. 1859-1868

2018

  1. Mechanisms of bacterial tolerance and persistence in the gastrointestinal and respiratory environments

    Clinical Microbiology Reviews, Vol. 31, Núm. 4

2016

  1. Toxin-antitoxin systems in clinical pathogens

    Toxins, Vol. 8, Núm. 7

2014

  1. Rare variants in PPARG with decreased activity in adipocyte differentiation are associated with increased risk of type 2 diabetes

    Proceedings of the National Academy of Sciences of the United States of America, Vol. 111, Núm. 36, pp. 13127-13132

2013

  1. Mutations in SLC20A2 are a major cause of familial idiopathic basal ganglia calcification

    Neurogenetics, Vol. 14, Núm. 1, pp. 11-22

2009

  1. Planning the human variome project: The Spain report

    Human Mutation, Vol. 30, Núm. 4, pp. 496-510